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    Interstitial Lung Disease Medical Slides

    Generate publication-quality interstitial lung disease lecture slides in 30 seconds. AI-powered content structured for clinical education.

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    Why teach Interstitial Lung Disease?

    Interstitial lung diseases encompass over 200 distinct entities with a combined prevalence of approximately 80 per 100,000. The 2022 ATS/ERS/JRS/ALAT guidelines emphasize multidisciplinary discussion (MDD) as the diagnostic gold standard, integrating clinical, radiological, and pathological data. Teaching ILD requires systematic HRCT pattern recognition and understanding of the diagnostic framework that guides management decisions.

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    What AI generates for Interstitial Lung Disease

    Enter “Interstitial Lung Disease” and SlideCraft generates a complete lecture deck with slides like these.

    01ILD Classification: Idiopathic Interstitial Pneumonias, CTD-ILD, and Exposure-Related
    02HRCT Patterns: UIP, NSIP, Organizing Pneumonia, and Lymphocytic Interstitial Pneumonia
    03Multidisciplinary Discussion: Integrating Clinical, Radiological, and Pathological Data
    04Pulmonary Function Testing: Restrictive Pattern, DLCO Reduction, and 6-Minute Walk
    05Antifibrotic Therapy: Pirfenidone and Nintedanib Indications Beyond IPF
    06Autoimmune Screening Panel: ANA, RF, Anti-CCP, Myositis-Specific Antibodies
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    Interstitial Lung Disease Presentation FAQ

    How should HRCT pattern recognition be taught in ILD slides?

    Present the four key patterns with characteristic features: UIP (basal-predominant honeycombing, traction bronchiectasis, minimal ground-glass), NSIP (basal ground-glass with subpleural sparing), OP (consolidation in peribronchial distribution, reversed halo sign), and HP (upper/mid-zone mosaic attenuation with air trapping). Emphasize that a definite UIP pattern on HRCT has >90% positive predictive value for histological UIP, often avoiding surgical biopsy.

    What serological workup should be highlighted for ILD evaluation?

    Present the systematic screening panel: ANA (CTD screen), RF and anti-CCP (rheumatoid arthritis), anti-Scl-70 and anti-centromere (scleroderma), anti-Jo-1 and other myositis-specific antibodies (antisynthetase syndrome), ANCA (vasculitis). Include serum KL-6 and SP-D as ILD activity biomarkers. Emphasize that up to 15% of apparently idiopathic ILD is reclassified as CTD-ILD after comprehensive autoimmune evaluation.

    How should antifibrotic therapy evidence be presented?

    Reference the pivotal trials: pirfenidone (ASCEND, 2014) reduced FVC decline by 50% in IPF; nintedanib (INPULSIS, 2014) reduced FVC decline by 50% in IPF. Present the expanding indications — nintedanib for progressive fibrosing ILD (INBUILD trial, 2019) and SSc-ILD (SENSCIS trial). Discuss the 2022 guidelines recommending antifibrotics for progressive pulmonary fibrosis regardless of underlying ILD diagnosis.

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