Electrical Injury Medical Slides
Generate publication-quality electrical injury lecture slides in 30 seconds. AI-powered content structured for clinical education.
Generate Electrical Injury DeckWhy teach Electrical Injury?
Electrical injuries cause approximately 1,000 deaths and 30,000 non-fatal injuries annually in the United States. High-voltage injuries (>1000V) cause devastating thermal tissue destruction along the current path, while low-voltage injuries primarily risk cardiac arrhythmias. Lightning strikes represent a unique mechanism with 10% mortality. The management requires understanding of current pathophysiology, recognition of occult internal injuries, and appropriate cardiac monitoring duration — all of which distinguish electrical from other burn injuries.
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Electrical Injury Presentation FAQ
How should cardiac monitoring duration be taught for electrical injuries?
Present the evidence-based approach: Low-voltage (<1000V) with normal ECG, no LOC, no cardiac symptoms → observe 4-6 hours, discharge if asymptomatic with normal repeat ECG. Low-voltage with ANY abnormality (arrhythmia, LOC, abnormal ECG, chest pain) → 24-hour telemetry monitoring. High-voltage (≥1000V) → minimum 24-hour monitoring regardless of initial ECG (delayed arrhythmias reported). Lightning strike → 24-hour monitoring (cardiac arrest at scene is common — VF or asystole). Troponin should be checked in all high-voltage and symptomatic low-voltage injuries. Late arrhythmias after initially normal 24-hour monitoring are exceedingly rare.
What internal injury assessment must be highlighted?
Present the "iceberg" concept: external burns dramatically underestimate internal injury in high-voltage cases — current travels along neurovascular bundles, causing deep tissue necrosis not visible on surface. Key assessments: (1) Rhabdomyolysis — CK levels (can exceed 100,000), tea/cola-colored urine, aggressive hydration to maintain UOP >1-2 mL/kg/hr, target CK <5000 before stopping aggressive fluids. (2) Compartment syndrome — high clinical suspicion in any extremity traversed by current, check pressures, early fasciotomy. (3) Vascular injury — arterial thrombosis from intimal damage. (4) Visceral injury — small bowel perforation, solid organ necrosis (rare but described with abdominal current path). Serial exams over 48-72 hours are essential as necrosis evolves.
How should lightning-specific management be taught?
Present the unique aspects of lightning injury: mechanism differs from industrial electrical injury — massive DC current with extremely brief duration (1-5 ms), flows primarily over the body surface (flashover effect). Reversed triage principle: resuscitate apparently dead victims first (most common cause of death is cardiac arrest — VF or asystole — with good neurological recovery if resuscitated promptly, because lightning often causes transient primary cardiac arrest rather than progressive multiorgan failure). Unique injuries: Lichtenberg figures (fernlike skin markings, pathognomonic, fade in hours), tympanic membrane rupture (50%), fixed dilated pupils (do NOT use as prognostic sign — may be transient from autonomic dysfunction).
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