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    Hypertensive Emergency Medical Slides

    Generate publication-quality hypertensive emergency lecture slides in 30 seconds. AI-powered content structured for clinical education.

    Generate Hypertensive Emergency Deck

    Why teach Hypertensive Emergency?

    Hypertensive emergency is defined as severe blood pressure elevation (typically >180/120 mmHg) with acute end-organ damage including hypertensive encephalopathy, aortic dissection, acute heart failure, or acute kidney injury. It accounts for approximately 1-2% of all emergency department visits for hypertension. Teaching requires distinguishing emergency from urgency and selecting targeted IV antihypertensive therapy based on the specific organ system involved.

    Sample Lecture Slides

    What AI generates for Hypertensive Emergency

    Enter “Hypertensive Emergency” and SlideCraft generates a complete lecture deck with slides like these.

    01Hypertensive Emergency vs Urgency: Defining End-Organ Damage
    02Target Organ Assessment: Brain, Heart, Kidney, Eyes, and Aorta
    03Blood Pressure Lowering Targets: The 25% Rule and Exceptions
    04IV Antihypertensive Selection: Nicardipine, Labetalol, Nitroprusside, and Clevidipine
    05Special Scenarios: Aortic Dissection, Eclampsia, and Pheochromocytoma Crisis
    06Transition to Oral Therapy and Outpatient Follow-Up Planning
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    Hypertensive Emergency Presentation FAQ

    How should blood pressure reduction targets be presented in hypertensive emergency slides?

    Teach the general rule: reduce MAP by no more than 25% in the first hour, then to 160/100 mmHg over the next 2-6 hours, then gradually to normal over 24-48 hours. Emphasize the critical exception: aortic dissection requires SBP <120 mmHg and HR <60 bpm within 20 minutes using IV esmolol or labetalol before adding vasodilators to prevent reflex tachycardia.

    What IV antihypertensive comparison should be included?

    Create a comparison table of nicardipine (titratable, no reflex tachycardia, preferred in stroke), labetalol (combined alpha/beta blockade, safe in pregnancy), clevidipine (ultra-short acting, lipid emulsion vehicle), and nitroprusside (cyanide toxicity risk, last resort). Reference the 2017 ACC/AHA Hypertension guidelines and the CLUE trial comparing clevidipine to nicardipine.

    How should posterior reversible encephalopathy syndrome (PRES) be addressed?

    Include PRES as a key teaching point in hypertensive encephalopathy. Show characteristic MRI findings of bilateral parieto-occipital vasogenic edema on FLAIR sequences. Explain the pathophysiology of failed cerebral autoregulation, emphasize that PRES is reversible with prompt BP control, and note that it can also occur with eclampsia, immunosuppressants (cyclosporine, tacrolimus), and cytotoxic chemotherapy.

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